2019 |
NGL-1/LRRC4C-mutant mice display hyperactivity and anxiolytic-like behavior associated with widespread suppression of neuronal activity. Choi Y*, Park H*, Kang S, Kweon H, Kim S, Lee SY, Jung H, Choi EY, and Kim E. [Front Cell Neurosci 12, 250-250(2019)] Abstract |
2019 |
Shank3 exons 14–16 deletion in glutamatergic neurons leads to social and repetitive behavioral deficits associated with increased cortical layer 2/3 neuronal excitability. Yoo T, Cho H, Park H, Lee J, and Kim E. [Front Cell Neurosci 13, 458-458(2019)] Abstract |
2019 |
A TBR1-K228E mutation induces Tbr1 upregulation, altered cortical distribution of interneurons, increased inhibitory synaptic transmission, and autistic-like behavioral deficits in mice. Yook C, Kim K, Kim D, Kang H, Kim SG, Kim E, and Kim SY. [Front Mol Neurosci 12, 241-241(2019)] Abstract |
2019 |
SynGO: an evidence-based, expert-curated resource for synapse function and gene enrichment studies. Koopmans F, van Nierop P...Kim E...,Verhage M. [Neuron 103(2), 217-234(2019)] Abstract |
2019 |
Shank3 mice carrying the human Q321R mutation display enhanced self-grooming, abnormal electroencephalogram patterns, and suppressed neuronal excitability and seizure susceptibility. Yoo YE, Yoo T, Lee S, Lee J, Kim D, Han HM, Bae YC, Kim E. [Front Mol Neurosci 12, 155-155(2019)] Abstract |
2019 |
Scn2a haploinsufficiency in mice suppresses hippocampal neuronal excitability, excitatory synaptic drive, and long-term potentiation, and spatial learning and memory. Shin W*, Kweon H*, Kang R*, Kim D, Kim K, Kang M, Kim SY, Hwang SN, Kim JY, Yang E, Kim H, and Kim E. [Front Mol Neurosci 12, 145-145(2019)] Abstract |
2019 |
NGL-3 in the regulation of brain development, Akt/GSK3b signaling, long-term depression, and locomotive and cognitive behaviors. Lee H*, Shin W*, Kim K*, Lee S, Lee EJ, Kim J, Kweon H, Lee E, Park H, Kang M, Yang E, Kim H, and Kim E. [PLoS Biol 17(6), e2005326-e2005326(2019)] Abstract |
2019 |
NGL-1/LRRC4C deletion moderately suppresses hippocampal excitatory synapse development and function in an input-independent manner. Choi Y*, Park H*, Jung H, Kweon H, Kim S, Lee SY, Han H, Cho Y, Kim S, Sim WS, Kim J, Bae Y, and Kim E. [Front Mol Neurosci 12, 119-119(2019)] Abstract |